Fanconi and inappropriate secretion of antidiuretic hormone syndromes secondary to venlafaxine therapy.
نویسندگان
چکیده
Accessible online at: www.karger.com/journals/nef Venlafaxine, a 2-phenyl-2 (1-hydroxycycloalkyl) ethylamine derivate, is an inhibitor of both serotonin and norepinephrine re-uptake and was termed a ‘dual uptake inhibitor’ [1]. We report on a venlafaxine-treated patient who developed Fanconi’s syndrome (FS) and hyponatremia secondary to inappropriate antidiuretic hormone secretion syndrome (SIADH).
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ورودعنوان ژورنال:
- Nephron
دوره 92 4 شماره
صفحات -
تاریخ انتشار 2002